Search Results for coronavirus

Claim that hydroxychloroquine-azithromycin improves COVID-19 recovery is unsupported at this stage

Clinical trials adhere to strict rules of design and execution. While abiding by these rules calls for lengthier or more complex trials, these rules are necessary to produce reliable results that can be used to conclusively identify effective therapies. Among the important rules of the scientific method, control groups are essential in order to provide an objective reference point. Contrary to numerous sensationalized headlines, many recent studies on the effects of hydroxychloroquine used alone or in combination with other drugs to treat COVID-19 have been inconclusive due to flawed study design.

Little to no evidence that smoking protects against COVID-19; may increase risk of complications instead

Preliminary studies showing a disproportionately low percentage of smokers among COVID-19 patients have led to suggestions that smoking protects against COVID-19. However, these studies have not demonstrated a causal association between smoking and a reduced risk of either SARS-CoV-2 infection or COVID-19 disease severity. Much more research is needed before any causal association can be established. Furthermore, the established science indicates that smoking increases the risk of developing respiratory infections and complications in general.

Eating alkaline food cannot prevent or cure COVID-19

Scientists have not determined the ideal pH environment for SARS-CoV-2. In addition, the cells of the tissues that SARS-CoV-2 primarily targets, including the lungs and intestines, maintain a very tightly controlled pH both inside and out in order to maintain cellular metabolism and proper enzyme activity. Altering pH in cells and tissues beyond a very narrow range would likely result in death.

Claim that SARS-CoV-2 binds to hemoglobin in red blood cells unsupported and implausible

The claim that COVID-19 causes hypoxia because the causative virus binds to hemoglobin in red blood cells is unsupported. Firstly, no scientific evidence demonstrates that SARS-CoV-2 can enter red blood cells. Secondly, the claim that the virus binds to hemoglobin is founded on the conclusions of a single pre-print, which solely involves computational analysis, without experimental verification or peer review. The mechanism proposed is also inconsistent with clinical evidence from COVID-19 patients.

Some evidence suggests that vitamin C and D supplementation might prevent or treat respiratory infections, but their effectiveness is still being tested with COVID-19

Although no public health agencies have recommended vitamins C or D as standard COVID-19 treatments, both have demonstrated some ability to shorten the length of respiratory infections by boosting the strength of the immune system. Vitamin D has also demonstrated the ability to reduce the risk of respiratory infection, especially in individuals who are vitamin D deficient. Many in the scientific and medical communities are recommending vitamin D supplementation or getting at least 15 minutes of sun exposure a day to prevent deficiencies.

Conspiracy theorists claim that 5G increases vulnerability to COVID-19 with baseless theory that it affects hemoglobin

Wireless communications use electromagnetic waves to transfer data. Electromagnetic waves carry energy that is proportional to their frequency and can interact with biological tissues. However, the energy level and the penetration depth of the type of waves used for 5G do not allow them to alter the structure and function of important biological macromolecules such as hemoglobin. In addition, there is no correlation between the very limited 5G coverage in the world and the progression of the COVID-19 pandemic.

COVID-19 vaccine candidate by Pittsburgh scientists show promising results in animal studies, but clinical trials still needed before efficacy in humans is known

Scientists who reviewed this article’s scientific credibility found that the reporting was generally accurate. However, they also highlighted a missed opportunity to provide readers with more information to place the research findings in context. Ian Frazer, immunologist and professor at the University of Queensland, pointed out that the article “assumes that an immune response in mice would equate to protection of humans against viral infection, but there are many examples where that is not the case”.